MAURI MARIO
Ruolo:
Tecnico - amministrativo
E-mail:
Pubblicazioni
- Karantzoulis, A., Conti, E., Slongo, M., Rodriguez-Menendez, V., Mauri, M., Da Re, F., et al. (2020). CLEARANCE OF BETA-AMYLOID OLIGOMERS BY PERIPHERAL MONOCYTES: FURTHER OBSERVATIONS REGARDING THE MODULATORY ROLE OF DONEPEZIL. Intervento presentato a: 51st Annual Conference of the Italian Society of Neurology, Virtual edition. Dettaglio
- Magistroni, V., Mauri, M., D'Aliberti, D., Mezzatesta, C., Crespiatico, I., Nava, M., et al. (2019). De novo UBE2A mutations are recurrently acquired during chronic myeloid leukemia progression and interfere with myeloid differentiation pathways. HAEMATOLOGICA, 104(9), 1789-1797. Dettaglio
- Ceccon, M., Mauri, M., Massimino, L., Giudici, G., Piazza, R., Gambacorti-Passerini, C., et al. (2018). Mitochondrial Hyperactivation and Enhanced ROS Production are Involved in Toxicity Induced by Oncogenic Kinases Over-Signaling. CANCERS, 10(12). Dettaglio
- Fontana, D., Mauri, M., Niro, A., Massimino, L., Bertagna, M., Zambrotta, G., et al. (2018). ETNK1 mutations promote ROS production and DNA damage through increased mitochondrial activity. In Proceedings: AACR Annual Meeting 2018. Dettaglio
- Fontana, D., Mauri, M., Niro, A., Massimino, L., Bertagna, M., Zambrotta, G., et al. (2018). ETNK1 MUTATIONS INCREASE MITOCHONDRIAL ACTIVITY AND PROMOTE DNA DAMAGE THROUGH ROS PRODUCTION. Intervento presentato a: XV Congress of the Italian Society of Experimental Hematology, Rimini, Italy. Dettaglio